I like features in the journals that keep you up to date with other literature, like the old annotated bibliography that AJNR used to have.  In that spirit, I will throw out some current literature that you might not have run across:

Occurrence of Basal ganglia germ cell tumors without a mass.  Arch Neurol  2009; 66(6): 789-792.  Kinda scary.  BG germ cell tumors, biopsy proven, presenting with volume loss and even wallerian degeneration.  Also hypometabolism on PET.

The timing and influence of MRI on the management of patients with cervical facet dislocations remains highly variable.  J Spinal Disord Tech 2009; 22(2): 96-99.  Title says it all.  No rhyme or reason to timing and use of MR.

Complications in the use of rhBMP-2 in PEEK cages for interbody spinal fusions.  J Spinal Disord Tech 2008; 21(8): 557-562.  Endplate resorption with BMP very common.  More likely to have graft migration with use of BMP in TLIF or PLIF without plate barrier.  See also letter response in volume 22, number 4, June 2009.

Interspinous process devices for the treatment of lumbar degenerative disease.  Current Orthopaedic Practice, 2009 ; 20(3): 232-237.  Nothing earth shattering, but a nice review.

Overdoseage of intrathecal gadolinium and neurolgical response.  Clinical Radiology  2008; 63: 1063-1068.  Why the heck would gadolinium be anywhere near the myelography room?  See also Journal of Neurology, 2007, Gadolinium encephalopathy due to accidental intrathecal administration of gadopentetate dimeglumine.  Always, always check the bottle of whatever you are injecting.  My first rule for all fellows and residents on myelography rotation.

MRI lesion profiles in sporadic Creutzfeldt-Jakob disease.  Neurology 2009; 72: 1994-2001.   Nice table showing the different diffusion appearances between the six molecular subtypes.    See also Ann Neurol 2000; 48:323-329 which kinda explains what the six molecular subtypes are.

Bilaterally symmetric form of Hirayama disease.  Neurology 2009; 72:2083-2089.  Anyone have a nice case of this?  Either a spinal muscle atrophy of unknown etiology, or related to cord compression from the posterior dura with flexion.  Still uncertain.  See also The Neurologist 2009; 15: 156-160  Familial Asymmetric distal upper limb amyotrophy (Hirayama Disease) : report of a Greek Family.