More recent various journal articles which may be of interest:
Deep Brain Stimulation, Neuroethics, and the minimally conscious state. Arch Neurol 2009; 66(6): 697-702. Case study of one patient who had bilateral thalamic electrodes, outlining the challenges and ethical implications.
Case 17-2009: A 30-year-old man with progressive neurological deficits. NEJM 2009; 360:2341-51. I will not give this one away, but I am adding this entity to my list of pathologies which can mimic anything (lymphoma, TB, sarcoid). Come on admit it, you go to the end and look at the answer first!
Primary angiitis of the central nervous system. Arch Neurol 2009; 66(6): 704-709. Good review with distinguishing features between it and reversible vasoconstriction syndrome. Regarding RVCS, see Ann Intern Med 2007; 146:34-44 for this excellent review.
Direct imaging of the distal dural ring and paraclinoid internal carotid artery aneurysms with high-resolution T2 turbo-spin echo technique at 3T magnetic resonance imaging. Neurosurgery 64:1059-1064, 2009. Nice to have another way to define the cave aneurysm, besides relation to ophthalmic artery and relation to optic strut.
Minimally invasive operative management for lumbar spinal stenosis: overview of early and long-term outcomes. Orthop Clin N Am 2007; 38: 387-399. More than you really want to know about this procedure, but useful to get an overview of endoscopic approaches for lumbar stenosis. Figure 9 summarized the whole article for me.
beta Amyloid, Blood vessels, and Brain Function. Stoke 2009; 40:2601-2606. Nice review of the cross-linking (pun intended) between cerebral amyloid angiopathy and Alzheimer’s. CAA is the most frequent vascular abnormality in Alzheimer’s.
CT angiography for intracerebral hemorrhage does not increase risk of acute nephropathy. Stroke 2009; 40:2393-2397. Large series where more patients got acute nephropathy who did NOT get CTA compared to those who did get CTA.
Brain lesions are more often reversible in acute thrombotic thrombocytopenic purpura. Neurology 2009; 73:66-70. Show interesting involvement of basal ganglia, which is apparently not just bilateral infarcts. They postulate as variant of PRES.
